Research
Cardiovascular disease
Free radical kinetics enable real-time detection of oxidative stress in cardiac cells, aiding early diagnosis and therapeutic monitoring of heart conditions.
Proof points
Hypoxia-reoxygenation: A rise in free radicals was detected during low-oxygen stress, followed by recovery with reoxygenation. Enables dynamic tracking of cardiac injury and recovery.
Shear stress: Blood flow changes caused a measurable increase in oxidative stress inside endothelial cells. Supports vascular health monitoring under physical or drug-induced stress.
Mitochondrial targeting: Real-time detection of mitochondrial free radicals at the subcellular level. Solves redox monitoring challenges in ischemia and drug assessment.
Advantages & complementarity
- Real-time oxidative stress quantification
- Subcellular resolution in living cells
- Complements cardiac imaging and troponin tests
- Detects early-stage redox imbalance
- Non-destructive, repeatable diagnostics
Partnership opportunities
We are actively seeking collaborations with researchers and clinical teams focused on heart failure, vascular dysfunction, or ischemia-reperfusion injury. We aim to integrate our free radical biomarker into clinical trials and therapeutic evaluations targeting oxidative stress and cardiovascular redox pathways.
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